Figure 4
(A) Western blot analysis of p-JNK, p-ERK1/2, p-p38, NF-κB-p65 and GRP78 in THP-1 cells treated with LPS and TM. (B) Western blot analysis of p-JNK, p-ERK1/2, p-p38, NF-κB-p65 and GRP78 in THP-1 cells treated with LPS and TG. GAPDH was loading control. Values were expressed as mean ± SD (n=5). *P<0.05 compared with untreated control cells. NS (not significant) compared with cells treated with LPS alone. #P<0.05 compared with cells treated with LPS alone.
p38 MAPK signaling contributed to TM or TG induced ER stress in THP-1 cells

(A) Western blot analysis of p-JNK, p-ERK1/2, p-p38, NF-κB-p65 and GRP78 in THP-1 cells treated with LPS and TM. (B) Western blot analysis of p-JNK, p-ERK1/2, p-p38, NF-κB-p65 and GRP78 in THP-1 cells treated with LPS and TG. GAPDH was loading control. Values were expressed as mean ± SD (n=5). *P<0.05 compared with untreated control cells. NS (not significant) compared with cells treated with LPS alone. #P<0.05 compared with cells treated with LPS alone.

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