Figure 5
(A) HepG2/Taxol cells treated with si-NC, si-NC+Taxol, si-HOTAIR, si-HOTAIR +Taxol, si-HOTAIR+Taxol+anti-miR-34a, anti-NC, anti-miR-34a and anti-miR-34a+Taxol. (B) SMMC7721/Taxol cells treated with si-NC, si-NC+Taxol, si-HOTAIR, si- HOTAIR +Taxol, si- HOTAIR+Taxol+anti-miR-34a, anti-NC, anti-miR-34a and anti-miR-34a+Taxol. The means of three independent biological replicates are shown; error bars indicate the SD. *P<0.05 vs. different controls.
Effect of HOTAIR and miR-34a on apoptosis rates in HepG2/Taxol and SMMC7721/Taxol cells

(A) HepG2/Taxol cells treated with si-NC, si-NC+Taxol, si-HOTAIR, si-HOTAIR +Taxol, si-HOTAIR+Taxol+anti-miR-34a, anti-NC, anti-miR-34a and anti-miR-34a+Taxol. (B) SMMC7721/Taxol cells treated with si-NC, si-NC+Taxol, si-HOTAIR, si- HOTAIR +Taxol, si- HOTAIR+Taxol+anti-miR-34a, anti-NC, anti-miR-34a and anti-miR-34a+Taxol. The means of three independent biological replicates are shown; error bars indicate the SD. *P<0.05 vs. different controls.

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