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Issues
December 2021
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Cover Image
Cover Image
Germline gain-of-function mutations in Protein Kinase C (PKC) are associated with neurodegenerative diseases. Aberrant PKC activity increases phosphorylation of its substrates to promote cytoskeleton disruption and neuronal death. Read about the repurposing of cancer drugs for neurodegenerative treatment in the review by Lordén and Newton in this issue of Neuronal Signaling (volume 5, issue 4).
EISSN 2059-6553
In this Issue
Review Articles
A sticky situation: regulation and function of protein palmitoylation with a spotlight on the axon and axon initial segment
Neuronal Signal (2021) 5 (4): NS20210005.
Conventional protein kinase C in the brain: repurposing cancer drugs for neurodegenerative treatment?
Neuronal Signal (2021) 5 (4): NS20210036.
3D hydrogel models of the neurovascular unit to investigate blood–brain barrier dysfunction
Neuronal Signal (2021) 5 (4): NS20210027.
Molecular mechanisms of action of stimulant novel psychoactive substances that target the high-affinity transporter for dopamine
Neuronal Signal (2021) 5 (4): NS20210006.
Mutations in DISC1 alter IP3R and voltage-gated Ca2+ channel functioning, implications for major mental illness
Neuronal Signal (2021) 5 (4): NS20180122.
Animal models of Parkinson’s disease: a guide to selecting the optimal model for your research
Neuronal Signal (2021) 5 (4): NS20210026.
Research Articles
Identification of TGFβ signaling as a regulator of interneuron neurogenesis in a human pluripotent stem cell model
Neuronal Signal (2021) 5 (4): NS20210020.