... Figure 2 MKP2 is up-regulated in TGF-β1-induced EMT The corrected Figure 2 is provided here: The authors of the original article, “MKP2 inhibits TGF-β1-induced epithelial-to-mesenchymal transition in renal tubular epithelial cells through a JNK-dependent pathway” (doi: 10.1042...

... inhibited LN metastasis. In conclusions, CEACAM5 inhibited epithelial–mesenchymal transition (EMT) in HNSCC by reducing murine double minute 2 (MDM2) expression and thereby suppressing LN metastasis. CEACAM5 has potential as both a prognostic marker and a therapeutic target in HNSCC. To knock down...

... change known as epithelial to mesenchymal transition (EMT). In this instance, cancer cells acquire migratory and invasive characteristics that allow the cells to move into adjacent tissue or enter the bloodstream to reach a secondary site, where they begin to form a new tumour. Targetting proteins...

...Zhenzhen Li; Xianghua Liu; Fengyan Tian; Ji Li; Qingwei Wang; Chaohui Gu Epithelial-to-mesenchymal transition (EMT) is a phenotypic conversion that plays a crucial role in renal fibrosis leading to chronic renal failure. Mitogen-activated protein kinase phosphatase 2 (MKP2) is a member of the dual...

... epithelial–mesenchymal transition (EMT) is the process through which epithelial cells switch phenotype towards cells with more mesenchymal features following epithelial damage and may occur in COPD, idiopathic pulmonary fibrosis and even lung cancer. In lung cancer, EMT is mainly thought to contribute...